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. 2017 Apr 29;18(5):942. doi: 10.3390/ijms18050942

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© 2017 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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RE-CE inhibits IR-induced apoptosis and DNA damage in LSKs. Mice were daily treated with vehicle or RE-CE as described in the Materials and Methods. (A) The percentage of apoptosis in LSKs; (B) The mean fluorescence intensity (MFI) of phospho-histone H2AX (γH2AX) in LSKs; (C) A representative FACS analysis of the percentage of cell apoptosis in LSKs; (D) A representative analysis of γH2AX expression in LSKs by flow cytometry. RE-CE treatment significantly reduced the percentage of cell apoptosis, and decreased the MFI of γH2AX in irradiated LSKs. All data are presented as means ± SEM (n = 5).