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. 2017 Apr 29;18(5):942. doi: 10.3390/ijms18050942

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© 2017 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Figure A1 — CE treatment ameliorates IR-induced injury in organ and peripheral blood. Mice were daily treated with the vehicle or CE as described in the Materials and Methods. (A) Representative H&E stained spleen, thymus, lung, and wright-giemsa stained peripheral blood （×40）on the 14th day after exposure to TBI; (B) The number of WBCs in peripheral blood 2 months after 6 Gy TBI; (C) Total splenocyte counts on the 14th day after exposure to TBI; (D) Total thymocyte counts on the 14th day after exposure to TBI.

Figure A1 — CE treatment ameliorates IR-induced injury in organ and peripheral blood. Mice were daily treated with the vehicle or CE as described in the Materials and Methods. (A) Representative H&E stained spleen, thymus, lung, and wright-giemsa stained peripheral blood （×40）on the 14th day after exposure to TBI; (B) The number of WBCs in peripheral blood 2 months after 6 Gy TBI; (C) Total splenocyte counts on the 14th day after exposure to TBI; (D) Total thymocyte counts on the 14th day after exposure to TBI.