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. 2017 Jun 2;7:2695. doi: 10.1038/s41598-017-02705-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Slight activation of β-catenin in the remodeling phases enhanced bone formation without obvious hindering bone resorption. (A–F) The ALP, OSX, Rux2, RANKL, OPG mRNA expression levels and RANKL/OPG ratio in WT, β-catenin-KO, CA-β-catenin and SA-β-catenin mice. (G–K) Representative TRAP staining of tibia fracture calluses in each group and the quantitative data. (L and M) The serum concentration of ALP and CTX in each group. The values are expressed as the means ± SDs. *p < 0.05, **p < 0.01 (Scale bar = 25 µm in G–J, n = 6/group).