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. 2017 Jun 5;14:114. doi: 10.1186/s12974-017-0885-7

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Experimental designs used in the current study. Workflow and treatment groups used to determine the effect of genetic deletion of PDE3A on inflammation-induced CMH development (a), pharmacological inhibition of PDE3A with cilostazol on CAA-associated spontaneous CMH development (b), and pharmacological inhibition of PDE3A with cilostazol on inflammation-induced CMH development (c)