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. 2017 Jun 5;8:131. doi: 10.1186/s13287-017-0575-5

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Involvement of HGF in the effect of ADHLSCs on HSC proliferation and procollagen type I secretion. a The numbers of floating and adhering HSCs after 24 h of incubation with ADHLSC- or HSC-conditioned medium was evaluated after pre-incubation with or without an anti-HGF antibody. There was significant, partial inhibition of the decrease in the number of adhering HSCs when incubated with ADHLSC-conditioned medium containing anti-HGF antibody (N > 5). b Inhibition of the effect of ADHLSC-conditioned medium on HSC pro-collagen type I secretion in the presence of an anti-HGF antibody (N = 3). c Anti-HGF antibody significantly inhibited the increase in HGF, IL-6, MMP1, and MMP2 secretion by HSCs. HGF hepatocyte growth factor, HSC hepatic stellate cells