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. 2017 Jun 8;7:3051. doi: 10.1038/s41598-017-03183-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of FA on the oxidative status and mitochondrial function in PBMCs and Thp-1 cells. FA increased MDA levels in both PBMCs and Thp-1 cells (A) had no effect on the mitochondrial membrane potential in PBMCs but increased depolarization of the mitochondrial membrane in Thp-1 cells (B). FA depleted ATP levels in PBMCs and Thp-1 cells (C). Data are expressed as mean ± SD. **p < 0.005 relative to respective control; ***p < 0.0001 relative to respective control.