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A model illustrating how LLG1 differentially regulates FLS2 accumulation, flg22-induced degradation, and signaling. (Left) In the wild-type plant, LLG1 is fully functional as a chaperone for FLS2. (Center) In the absence of the LLG1 chaperone, FLS2 manifests defects in accumulation, degradation, and signaling. (Right) In the llg1-3 mutant, the LLG1–3 variant retains chaperone function, but the conformation of LLG1–3 and FLS2 may be affected, which impairs FLS2 signaling.
