Table 6.
ROI | WM | GM | Neuronal activity | Aβ | Total |
---|---|---|---|---|---|
Frontal lobe | xxx | xxxxxxx | xx | 12 | |
Limbic system | xxx | xxxx | xxx | x | 11 |
Parietal lobe | xxxxxxxx | xx | x | 11 | |
Motor areas | xxx | xxxxx | xx | 10 | |
Temporal lobe | x | xxxx | x | x | 7 |
Cerebellum | xxxx | xx | 6 | ||
Basal ganglia | xxx | xx | 5 | ||
Occipital lobe | x | xx | x | 4 | |
Insula | xxx | 3 | |||
| |||||
Total by pathology type | 10 | 39 | 14 | 6 |
WM, white matter; GM, grey matter, Aβ, amyloid beta protein
It should be noted that Buchman and colleagues (2008; 2013) [66, 67] investigated the association between AD pathology (neuritic plaques, diffuse plaques, and neurofibrillary tangles) averaged across midfrontal, superiortemporal, inferior parietal, entorhinal, and hippocampal ROIs and gait, so it was not possible to differentiate by ROI or pathology type in these instances.