Table 1.

Patient characteristics, demographics and distribution of Status epilepticus subtypes

Variable	Value
(A) Clinical characteristics and distribution of SE subtypes among all included patients (n=275)^*
Presumed etiology of SE
AIS	9 (3)
Old stroke	26 (9)
Epilepsy	32 (12)
Brain tumor	30 (11)
Intracranial hemorrhage	26 (10)
Alcohol or drug withdrawal	18 (7)
Traumatic brain injury	16 (6)
Infectious encephalitis	16 (6)
Neurodegenerative disease	14 (5)
Post brain surgery	13 (4)
Acute metabolic derangement	12 (4)
Others	32 (12)
Unknown	31 (11)
Presumed etiology grouped according to the International League Against Epilepsy [4]
Acute etiology	124 (45)
Remote etiology	68 (25)
Progressive central nervous system disorders	23 (8)
Unprovoked/unknown etiology	60 (22)
SE types
NCSE in coma	87 (32)
Complex partial SE	113 (41)
Convulsive SE	58 (21)
Simple partial SE	13 (5)
Absence SE	4 (1)
(B) Demographics, baseline characteristics of patients with NCSE and AIS (n=9)^†
Age, median years (range)	75 (58–84)
Sex, female	5 (56)
Glucose, mmol/L, median (range)	7 (6–9.9)
Creatinine, mmol/L, median (range)	110 (43–428)
Modified Charlson Comorbidity Index, median (range)	2 (0–6)
History of prior ischemic stroke^‡	2 (22)
Lesion size on DWI-MRI)
Small: 1-10 cm³	4 (44)
Medium: 11-100 cm³	3 (33)
Large: >100 cm³	2 (22)
Cortical involvement	9 (100)

Values are presented as n (%) unless otherwise indicated.

SE, status epilepticus; AIS, acute ischemic strok; NSCE, nonconvulsive SE.

(A) Clinical characteristics and distribution of SE subtypes among all patients included in the study;

^†

(B) Baseline and neuroimaging characteristics of the nine patients with AIS in close temporal association to nonconvulsive SE (NCSE);

^‡

Prior ischemic stroke defined as: Prior acute focal neurological deficit(s) attributable to focal brain ischaemia, lasting >24 hours.