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. Author manuscript; available in PMC: 2018 Jul 1.
Published in final edited form as: Cell Microbiol. 2017 Feb 20;19(7):10.1111/cmi.12728. doi: 10.1111/cmi.12728

Figure 6.

Figure 6

HhCDT-deficiency decreased the potential of H. hepaticus to induce activation of Stat3 at 20 WPI. (A) The percentage of the phospho-Stat3 foci+ cells in the cecal tissues from mice dosed with Hh, or the HhCDT mutant or vehicle alone at both 10 and 20 WPI. Hh-infected mice contained significantly more phospho-Stat3 foci+ cells in the cecum compared with the HhCDT mutant-infected mice at 20 WPI, but at 10 WPI. (B) The representative phospho-Stat3 staining images of the ICC/cecal tissue. (C) Linear regression analysis showing that there is the positive correlation between the numbers of the phospho-Stat3 foci+ cells and the scores of dysplasia. * represents P values when compared to the sham controls: ***<0.001, ****<0.0001.