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. 2017 May 31;13(5):e1006827. doi: 10.1371/journal.pgen.1006827

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© 2017 Su et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 4 — (A-L) Wild type (WT), atrad51^-/+ atrad51c^-/+ and atrad51^-/+ atxrcc3^-/+ mutant chromosome morphologies at pachytene, diakinesis, metaphase I and metaphase II. Compared with WT, atrad51^-/+ atrad51c^-/+ and atrad51^-/+ atxrcc3^-/+ mutants had similar chromosome morphologies. (M) atrad51c^-/+ atxrcc3^-/+ chromosome morphology at pachytene was similar to WT, but showed 32.8% (n = 61) non-homolog association at diakinesis (N). (O, P) atrad51c^-/+ atxrcc3^-/+ chromosome morphology at metaphase I and II, respectively. Chromosome morphology of atrad51^-/+ atrad51c^-/+ atxrcc3^-/+ at pachytene (Q) and diakinesis (R), metaphase I (S) and metaphase II (T). The atrad51^-/+ atrad51c^-/+ atxrcc3^-/+ triple chromosome morphology showed more severe defects than atrad51c^-/+ atxrcc3^-/+. Red dots indicate centromere signals. Yellow arrows show non-homologous chromosome associations (N, R, S). The red numbers in N, R, S refer to the number of abnormal cells out of all cells observed. Scale bar: 5 μm.