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. 2017 Jun 14;7:3494. doi: 10.1038/s41598-017-03796-5

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Plasma and hepatic lipids in hyperlipidemic mice with or without PepA-treatment. (A,B) Plasma total cholesterol and triglyceride measurements. (C,D) Total hepatic cholesterol and hepatic triglyceride measurements. (E) Representative pictures (original magnification, 200x) of the Oil red O staining. (F) Gene expression analysis of Cd36, Npc2, Lxrα and Ppar-γ. Gene expression data are shown relative to control mice on chow diet. Error bars represent ± SEM; *indicates p ≤ 0.05, **p < 0.01 and ***p < 0.001 compared to mice on chow diet; ^#indicates p ≤ 0.05, ^## p < 0.01 and ^### p < 0.001 compared to control-injected mice on HFC diet by use of two-tailed unpaired t test. n = 9–11 animals for each group analysis.