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. 2017 Jun 14;7:3448. doi: 10.1038/s41598-017-03449-7

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of miR-10b on proliferation in normal human organ-cultured corneas and HCEC. (a) miR-10b treatment in human organ-cultured corneas led to increased expression of Ki-67 in mimic-treated cornea compared to the fellow cornea treated with scrambled control, mimic control, or miR-10b inhibitor. (b) MTS proliferation assay showed significant increase or decrease in proliferation rates in HCEC transfected with miR-10b (M), or miR-10b inhibitor (I), respectively, in comparison to their respective transfected scrambled controls, mimic control (MC) and inhibitor control (IC). *P < 0.05, compared with scrambled control values by paired two-tailed t test.