Table 1B.

Echocardiographic parameters from control and fructose‐treated mice

	CD	FRD
n	9	13
LVDD (mm)	3.40 ± 0.11	3.77 ± 0.20
LVSD (mm)	2.40 ± 0.10	2.87 ± 0.14*
FS (%)	29.80 ± 2.35	23.93 ± 1.58*
LVMI (mg g−1)	7.90 ± 0.66	12.19 ± 1.19*
LVMI (mg mm−1)	14.10 ± 1.16	21.00 ± 2.43*

Diastolic left ventricular diameter (LVDD) was not different between the fructose‐rich diet (FRD) and control diet (CD) mice. However, left ventricular systolic diameter (LVSD) and the hypertrophy indices [LVMI: left ventricular mass index, normalized by body weight (mg g⁻¹) or by tibia length (mg mm⁻¹)] were significantly increased in FRD with respect to CD animals. These altered parameters, in association with the fractional shortening (FS: endocardial fractional shortening) being significantly decreased in FRD vs. CD mice, support a cardiac dysfunction produced by the diet. Values are mean ± SEM. ^* P < 0.05 vs. control mice.