Skip to main content
. 2017 Apr 11;31(7):3150–3166. doi: 10.1096/fj.201601305R

TABLE 2.

Affinity and selectivity of existing clinically used and novel β-AR ligands as determined from antagonism of cimaterol-induced 3H-cAMP accumulation in CHO-β1, CHO-β2, or CHO-β3 cells

Compound Human β1-AR
Human β2-AR
Human β3-AR
β1 vs. β2 selectivity
Log Kd,β1 n Schild slope n Log Kd,β2 n Log Kd,β3 n Kd,β2/Kd,β1
Bisoprolol −8.60 ± 0.03 15 0.97 ± 0.03 5 −6.76 ± 0.03 13 −5.65 ± 0.06 5 69
Carvedilol −9.75 ± 0.12 12 1.29 ± 0.09 4 −10.62 ± 0.08 4 −8.54 ± 0.18 3 0.13
Nebivolol −9.28 ± 0.08 22 1.15 ± 0.03 5 −7.91 ± 0.09 11 −6.35 ± 0.06 9 23
CGP20712A −9.64 ± 0.03 15 1.03 ± 0.03 5 −6.02 ± 0.03 8 −5.47 ± 0.04 5 4169
NDD-713 −8.52 ± 0.03 12 1.06 ± 0.03 4 −5.44 ± 0.03 4 −5.47 ± 0.08 3 1202
NDD-825 −8.99 ± 0.05 9 1.04 ± 0.02 3 −5.75 ± 0.06 4 −5.72 ± 0.08 7 1738

The Schild slope is that obtained in CHO-β1 cells from the rightward shift of the cimaterol dose-response curve by 3 different concentrations of antagonist in the same experiment (e.g., Fig. 3) where a value of 1 represents competitive antagonism. The affinity of most ligands at β2-AR and β3-AR was too low to allow a Schild plot to be constructed. Values shown are means ± sem of n separate experiments.