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. 2017 May 29;9:10–20. doi: 10.1016/j.dadm.2017.05.005

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© 2017 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

PMC Copyright notice

Fig. 3 — The proportion of Aβ positivity and topographical distribution of Aβ in frontotemporal dementia (FTD) cases with and without a genetic mutation. The proportion of patients that demonstrate no Aβ deposition (A0), Aβ in the frontal-temporal cortices (A1), additional Aβ in the basal ganglia (A2), and additional Aβ in the substantia nigra (A3) is shown in FTD cases without a mutation (n = 55), a MAPT mutation (n = 4), a C9ORF72 expansion (n = 10), and a GRN mutation (n = 7) is shown. As only one FTD case without a mutation had an A3 distribution (1%, 1/73), this has been combined with the A2 group.