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. 2017 Jun 16;12(6):e0179582. doi: 10.1371/journal.pone.0179582

Fig 2. Mutation or truncation of MinE’s membrane targeting sequence causes higher bulk stimulation of MinD’s ATPase activity, indicating increased antagonistic potential of MinE in the absence of its direct membrane interaction.

Fig 2

ATPase stimulation assay with WT MinE or MinE ∆(2–12), L3E, L4E, F6E, F7E using 4 μM MinD, 4 μM MinE and 0.2 mg/mL small unilamellar vesicles made of E. coli polar lipids. Error bars represent standard deviation from three independent experiments.