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. 2017 Jun 7;61(1):1330096. doi: 10.1080/16546628.2017.1330096

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© 2017 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 5. — Suppression by rosmarinic acid (RA) of lipopolysaccharide (LPS)-stimulated tumor necrosis factor-α (TNF-α) messenger RNA (mRNA) expression and secretion in RAW 264.7 macrophages, and inflammatory cytokine mRNA expression in 3T3-L1 adipocytes stimulated with LPS-macrophage-conditioned medium (MCM). RA inhibited LPS-stimulated TNF-α mRNA expression (A) and secretion (B) in RAW 264.7 macrophages. RA inhibited interleukin-6 (IL-6), IL-1β, monocyte chemoattractant protein-1 (MCP-1), and regulated on activation, normal T cell expressed and secreted (RANTES) mRNA expression in 3T3-L1 adipocytes incubated with LPS-MCM (C). *p < 0.05 vs vehicle (Veh) group; #p < 0.05 vs LPS group (in A and B); #p < 0.05 vs LPS-MCM group (in C).