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Brief summary of the metabolic networks involved in the effects of rosmarinic acid (RA) on adipogenesis and lipolysis. RA inhibits adipogenesis via phosphorylated extracellular signal-regulated kinase-1/2 (p-ERK1/2) and mothers against decapentaplegic homolog 3 (p-Smad3), basal lipolysis via phosphodiesterase-3 (PDE3), and isoproterenol (ISO)- and forskolin-stimulated lipolysis via hormone-sensitive lipase (p-HSL) and p-perilipin A.
