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. 2017 Jun 14;10:2993–2999. doi: 10.2147/OTT.S136218

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© 2017 Yang et al. This work is published and licensed by Dove Medical Press Limited

The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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The effect of 5-AZA and TSA on the growth and viability of human lung cancer cells’ growth and viability.

Notes: (A) 5-AZA inhibited the viability of NCI-H1975 and NCI-H1299 cells at days 2 and 3. Cells were treated with 0, 0.1, 1, 5, 10, and 20 µM 5-AZA. (B) 5-AZA inhibited the proliferation of NCI-H1975 and NCI-H1299 cells at day 3. (C) TSA decreased the viability of NCI-H1975 and NCI-H1299 cells at day 2. (D) TSA decreased the growth of NCI-H1975 and NCI-H1299 cells at days 2 and 3. (E) The joint application of 5-AZA and TSA significantly inhibited the viability of NCI-H1975 and NCI-H1299 cells. *P<0.05, **P<0.01. N=3.

Abbreviations: 5-AZA, azacitidine; Rel, relative; TSA, trichostatin A.