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. 2017 May 4;12(5):734–743. doi: 10.2215/CJN.10601016

Table 4.

Multivariable prediction model for the course of eGFR during follow-upa

Model ISN/RPS Class I–V (n=105) ISN/RPS Class III/IV (±V)b (n=91)
β (95% CI) P Value β (95% CI) P Value
Intercept 116.5 (100.1 to 132.8) <0.001 116.4 (98.9 to 133.9) <0.001
Time, yr −0.7 (−1.5 to 0.0) 0.06 −0.6 (−1.4 to 0.2) 0.13
Baseline predictors
 Nonwhite −11.4 (−21.9 to −0.8) 0.04 −13.5 (−25.2 to −1.7) 0.03
 Age0, yr −0.8 (−1.2 to −0.4) <0.001 −0.8 (−1.2 to −0.4) <0.001
 Normal glomeruli/minimal leukocyte influx, % 0.2 (0.1 to 0.4) 0.01 0.2 (0.0 to 0.5) 0.03
 Cellular/fibrocellular crescents, % −0.4 (−0.6 to −0.2) 0.001 −0.4 (−0.6 to −0.1) 0.003
 Fibrous crescents, % −1.4 (−2.4 to −0.5) 0.004 −1.6 (−2.6 to −0.5) 0.004
 IF/TA≥25% −40.5 (−56.2 to −24.8) <0.001 −41.4 (−58.3 to −24.4) <0.001

β indicates eGFR in ml/min per 1.73 m2. eGFR at time t is given by the following: eGFR(t) = intercept + βTime×t + Z, where Z is the value given by the baseline predictors of the patient: Z = βAge0 × Age0[y] + βNormal glomeruli/minimal leukocyte influx × Normal glomeruli/minimal leukocyte influx[%] + βcellular/fibrocellular crescents × cellular/fibrocellular crescents[%] + βfibrous crescents × fibrous crescents[%] + βnonwhite (if nonwhite) + βIF/TA≥25% (if IF/TA≥25%). ISN/RPS, International Society of Nephrology/Renal Pathology Society; 95% CI, 95% confidence interval; Age0, age at time of biopsy; IF/TA, interstitial fibrosis/tubular atrophy.

a

Mixed-model analysis.

b

Patients with class III/IV (±V) lupus nephritis who received induction immunosuppression with cytotoxic drugs were analyzed separately from the complete cohort.