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. 2017 Apr 6;8(4):e2735. doi: 10.1038/cddis.2017.90

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Copyright © 2017 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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IL-33-MyD88 signaling in DCs is required for AD development. (a) DC infiltration in skin lesions. (b–d) The ear thickness (b), clinical score (c) and H&E staining of the ear (d) of BL6 and MyD88-floxed × CD11c-cre mice after 12 days of MC903 treatment. Scale bar represents 50 μm. (e-i). The protein expression of MPO (e), MMP-9 (f), LCN-2 (g), eotaxin-2 (h), IL-4 (i), IL-5 (j) and IL-13 (k) in the ear of BL6 and MyD88-floxed × CD11c-cre mice treated with ethanol or MC903 at day 12. *P<0.05,**P<0.01,***P<0.001.P-values were analyzed by two-tailed t-tests in a and one-way ANOVA in b-k. All data are representative of two to three independent experiments with n=7-8 per group and are means±S.E.M.