Table 1.
List of KEGG pathways enriched in SGBS adipocytes when compared to PHWSC adipocytes.
| KEGG biological pathways | FDR |
|---|---|
| ECM-receptor interaction | 3.18E-06 |
| Metabolic pathways | 3.07E-05 |
| Type I diabetes mellitus | 0.000142 |
| Fatty acid metabolism | 0.000178 |
| Cytokine-cytokine receptor interaction | 0.000256 |
| Calcium signaling pathway | 0.000443 |
| Autoimmune thyroid disease | 0.001638 |
| PI3K-Akt signaling pathway | 0.002073 |
| Neuroactive ligand-receptor interaction | 0.003842 |
| Pathways in cancer | 0.004007 |
| Hematopoietic cell lineage | 0.004051 |
| Graft-versus-host disease | 0.005511 |
| Cell adhesion molecules (CAMs) | 0.005968 |
| Fatty acid degradation | 0.006437 |
| Biosynthesis of unsaturated fatty acids | 0.009968 |
| Valine, leucine and isoleucine degradation | 0.013166 |
| Focal adhesion | 0.016408 |
| Allograft rejection | 0.018391 |
| PPAR signaling pathway | 0.029223 |
| Glycerolipid metabolism | 0.044868 |
Genes with log (FC) > 0.60 and FDR < 0.05 were used as an input in ipathway guide to generate significantly enriched KEGG pathways (P < 0.05, Bonferonni corrected). Pathways are listed in increasing order of P value.