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. Author manuscript; available in PMC: 2018 Jul 1.
Published in final edited form as: Neurobiol Dis. 2017 Apr 2;103:45–53. doi: 10.1016/j.nbd.2017.03.017

Figure 3. Phosphorylation of S831 and S845 residues of the GluA1 subunit was reduced in animals in SE.

Figure 3

(A) The expression of the GluA1 subunit phosphorylated on residues S831 and S845 in proteins isolated from the CA1 area of SE (40 min after the first stage 5 behavioral seizures induced by pilocarpine) and naïve animals. The expression of β-actin was determined in each gel for normalization. The expression of the GluA1 subunit was also determined in each sample. (B) The expression of pGluA1 and total GluA1 subunit was normalized to that of β-actin and presented relative to that in naïve animals (N= 5, * p<0.005, Wilcoxon matched-pairs signed-rank test).