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. 2017 May 17;16(1):133–142. doi: 10.3892/mmr.2017.6591

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Copyright: © Kim et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — SOD2 immunohistochemistry in the hippocampal CA1 region of the (A) vehicle-sham-, (B) CIL-sham-, (C and E) vehicle-ischemia- and (D and F) CIL-ischemia-groups following ischemia-reperfusion. SOD2 immunoreactivity is identified in the SP in the vehicle-sham-group, and SOD2 immunoreactivity in the SP (asterisk) is barely observable 5 days following ischemia-reperfusion. In the CIL-sham-group, SOD2 immunoreactivity is similar to that of the vehicle-sham-group, and, in the CIL-ischemia-group, SOD2 immunoreactivity in the SP is higher than that corresponding vehicle-sham-group. Scale bar, 100 µm. (G) ROD as % values of SOD2 immunoreactivity in the SP of each group (*P<0.05 vs. vehicle-sham-group; ^#P<0.05 vs. corresponding vehicle-ischemia-group; ^†P<0.05 vs. respective pre-time point group). The bars indicate the means ± standard error of mean. SP, stratum pyramidale; SOD, superoxide dismutase; CIL, Chrysanthemum indicum Linné extract; ROD, relative optical density; SO, stratum oriens; SR, stratum radiatum.