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. 2017 May 16;16(1):109–118. doi: 10.3892/mmr.2017.6586

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Octreotide promotes the phosphorylation of Akt and GSK3β and the expression of GS mRNA in rats with HFD-induced obesity. (A) Western blotting images of p-Akt (Ser473) and (B) quantification of band intensities. (C) Western blotting images of p-GSK3β (Ser9) and (D) quantification of band intensities. (E) The level of GS mRNA. Data presented as the mean ± standard deviation of three independent experiments. *P<0.05 vs. control group; ^#P<0.05 vs. HFD group. p-Akt, phosphorylated-Akt; p-GSK3β, phosphorylated-glycogen synthase kinase 3β; GS, glycogen synthase; HFD, high fat diet; IOD, integrated optical density.