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. 2017 Jun 27;18:104. doi: 10.1186/s12931-017-0591-0

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — In utero SHS exposure dysregulates the expression of 33 genes, including functional clusters related to fibronectin, kinase and transcription regulation in 15-week old male mice. Results are presented for global gene expression of left lung (see methods for details regarding the gene expression analysis). Data are expressed as in utero SHS-exposed mice compared with air-exposed controls (log2 ratio fold increases) (n = 4 mice per group). Genes with at least 2-fold up-/down-regulation (p < 0.05) and FDR < 0.05 were considered differentially expressed