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. 2017 May 16;14(1):830–836. doi: 10.3892/ol.2017.6179

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Copyright: © He et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Effects of miR-106a on apoptosis of colorectal cancer cells. HCT116 cells were transfected with (A) mimic control or (B) miR-106a mimic, and (C) the proportion of cells in early apoptosis was quantified. HCT116 cells were transfected with (D) inhibitor control or (E) miR-106a inhibitor mimic, and (F) the proportion of cells in early apoptosis was quantified. Results are representative of at least 3 independent experiments. P-values were calculated with an unpaired t-test and are expressed in comparison with the respective controls. miR-106a, microRNA-106a; FITC, fluorescein isothiocyanate.