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. 2017 Jul 3;19:79. doi: 10.1186/s13058-017-0872-z

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Loss of ERβ is associated with occurrence of EMT in mammary tumors. Microphotographs of serial tumor sections analyzed by immunohistochemistry for the expression of E-cadherin, vimentin, α-SMA and the cytoplasmic active form of B-catenin. Areas of tumors arising in the absence of ERβ (K14CreERβ ^F/F p53 ^F/F mice) display decreased levels or loss of E-cadherin and increased expression of vimentin, α-SMA and B-catenin. Scale bars, 100 μm. ER estrogen receptor, α-SMA alpha-smooth muscle actin