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. 2017 Jun 30;8:15921. doi: 10.1038/ncomms15921

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Copyright © 2017, The Author(s)

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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DS events in AA PCa versus EA PCa were frequently detected in multiple oncogenic signalling pathways. Depicted pathway is a composite of PI3K/AKT/mTOR, RAS/RAF/MAPK, CDK/RB1, MDM2/TP53 and WNT/GSK3/CTNNB1/APC signalling. Open figures indicate oncogenes and closed figures indicate tumour suppressor genes. ‘DS’ indicates that a differential alternative splicing event was detected for a particular oncogene or tumour suppressor gene.