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. 2017 Jul 6;18:517. doi: 10.1186/s12864-017-3895-z

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Histology of gastric carcinomas with GNAS-mutations (patients No. 3–6 and 9). Seven different gastric cancers with mutations in exon 8 of GNAS showed different phenotypes (a-g). GNAS was detected by immunohistochemistry in 41 gastric cancer specimens and showed membranous and weak cytoplasmic staining. H&E-staining (a-e); anti-GNAS-immunostaining (f). Original magnifications 200×