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. 2017 Apr 8;32(5):431–441. doi: 10.1007/s10654-017-0244-0

Table 3.

Genetic variants showing a clear dose–response relationship in their association with ovarian cancer risk, 20.3 years of follow-up

Main effects SNPs Homozygous wildtype 1 or 2 variant alleles 1 variant allele 2 variant alleles p trend per allele Benjamini–Hochberg-adjusted p value
N cases HR (95% CI)a N cases HR (95% CI)a N cases HR (95% CI)a N cases HR (95% CI)a
CAT, rs511895 86 Ref 215 1.25 (0.95–1.63) 154 1.17 (0.88–1.56) 61 1.48 (1.04–2.13) 0.04 0.59
CYP1A1, rs4646903 261 Ref 36 0.70 (0.48–1.02) 36 0.70 (0.48–1.02) na 0.06 0.59
CYP1B1, rs1056827 144 Ref 154 1.26 (0.99–1.62) 127 1.24 (0.96–1.61) 27 1.36 (0.87–2.14) 0.06 0.59
MUTYH, rs3219489 189 Ref 112 0.78 (0.60–1.00) 97 0.79 (0.61–1.03) 15 0.70 (0.40–1.23) 0.05 0.59
TXN, rs2301241 95 Ref 206 1.26 (0.97–1.65) 147 1.18 (0.89–1.56) 59 1.55 (1.08–2.22) 0.02 0.59
GSTM1 deletion 1 or 2 alleles present Homozygous deletion p value Benjamini–Hochberg-adjusted p value
N cases HR (95% CI)a N cases HR (95% CI)a
Deletion represented by
 Both GSTM1 SNPs 226 Ref 75 0.74 (0.56–0.98) 0.03 0.59
 rs10857795 214 Ref 87 0.73 (0.56–0.95) 0.02 0.59
 rs200184852 185 Ref 116 0.84 (0.66–1.09) 0.19 0.59

aHR (95% CI): hazard ratio with corresponding 95% confidence interval; hazard ratios are adjusted for age; na not applicable