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. 2017 Jul 10;12(7):e0180712. doi: 10.1371/journal.pone.0180712

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© 2017 Chou et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 4 — Effects of aliskiren on markers of oxidative stress in visceral adipose tissues, namely (A) superoxide dismutase (SOD) and (B) thiobarbituric acid reactive substances (TBARS), in fructose-fed hypertensive rats. Con: control rats were fed the normal chow diet; Fru: rats were fed the high-fructose diet for 8 weeks; FruA: rats received the same treatment as Group Fru, and aliskiren was concurrently administered; FruB: rats received the same treatment as Group Fru, and aliskiren was administered 4 weeks after the initiation of high-fructose feeding. Values are expressed as means ± SD mean of six independent samples. ^ and # denote P < 0.05 versus control rats and fructose-fed rats, respectively. N = 6 per group.