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. 2017 Jul 17;8:798. doi: 10.3389/fimmu.2017.00798

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Copyright © 2017 Dobbs, Tabellini, Calzoni, Patrizi, Martinez, Giliani, Moratto, Al-Herz, Cancrini, Cowan, Bleesing, Booth, Buchbinder, Burns, Chatila, Chou, Daza-Cajigal, Ott de Bruin, de la Morena, Di Matteo, Finocchi, Geha, Goyal, Hayward, Holland, Huang, Kanariou, King, Kaplan, Kleva, Kuijpers, Lee, Lougaris, Massaad, Meyts, Morsheimer, Neven, Pai, Parvaneh, Plebani, Prockop, Reisli, Soh, Somech, Torgerson, Kim, Walter, Gennery, Keles, Manis, Marcenaro, Moretta, Parolini and Notarangelo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Flow cytometry analysis of the expression of CD57 (A,B), NKG2A (C,D), and CXCR1 (E,F), and killer cell immunoglobulin-like receptors (KIRs) (G,H) among CD3⁻ CD19⁻ CD14⁻ CD56⁺ cells. In panels (A,C,E,G), representative examples are shown for one patient each belonging to the severe combined immune deficiency (SCID), Omenn syndrome (OS), and delayed-onset combined immune deficiency (CID) subgroups. For each marker, positivity was defined as fluorescence intensity above that of isotype control. Bars represent mean ± SD values. *p < 0.05; **p < 0.01; ***p < 0.005; ****p < 0.001.