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. 2017 Jul 17;7:5567. doi: 10.1038/s41598-017-05856-2

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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HOXA11-AS RNAi promoted apoptosis in A549, H1299, H460 and PC9 cell lines based on a flow cytometry assay. (Note: control, lenti-control virus). (A) Comparison of the apoptosis rate and mortality rate of A549 cells after silencing HOXA11-AS expression (*P < 0.05, **P < 0.01, ***P < 0.001). (B) Comparison of the apoptosis rate and mortality rate of H460 cells after silencing HOXA11-AS expression (*P < 0.05, **P < 0.01, ***P < 0.001). (C) Comparison of the apoptosis rate and mortality rate of H1299 cells after silencing HOXA11-AS expression (*P < 0.05, **P < 0.01, ***P < 0.001). (D) Comparison of the apoptosis rate and mortality rate of PC9 cells after silencing HOXA11-AS expression (*P < 0.05, **P < 0.01, ***P < 0.001).