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. 2017 Jul 17;7:5518. doi: 10.1038/s41598-017-05791-2

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The modification effects of liver function on relationships of MetS and its constituents with HBV infection, Aflatoxin B1 exposure and MC-LR exposure. (a) The association between the liver function and HBV infection. (b) The association between the liver function and Aflatoxin B1 exposure. (c) The association between the liver function and MC-LR exposure. HBV infection and the higher AFB1 exposure (EDI) were inversely associated with MetS no matter whether the liver function was normal or abnormal. For people with HBV infection, the number of the constituents of MetS with decreased risk was higher in those showing abnormal liver function than in those showing normal liver function. For people with the higher AFB1 level, the number of the constituents of MetS with decreased risk were lower in those showing abnormal liver function than in those showing normal liver function. For people showing either normal or abnormal liver function, those exposed to the higher or lower MC-LR levels showed no difference in the risk of MetS and all its constituents.