Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2017 Jul 17;7:5518. doi: 10.1038/s41598-017-05791-2

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

The modification effects of HBV infection on relationships of MetS and its constituents with AFB1 exposure and MC-LR exposure; and the modification effects of MC-LR exposure on relationships of MetS and its constituents with AFB1 exposure. (a) The modification effects of HBV infection on the association between MetS and Aflatoxin B1 exposure. (b) The modification effects of HBV infection on the association between MetS and MC-LR exposure. (c) The modification effects of MC-LR exposure on the association between the MetS and Aflatoxin B1 exposure. The higher AFB1 exposure significantly decreased the risk of MetS no matter whether the HBV infection was positive or negative. For people with the higher AFB1 exposure, the number of the constituents of MetS with decreased risk was higher in those without HBV infection than in those with HBV infection. For people either with or without HBV infection, those exposed to the higher or lower MC-LR levels showed no difference in the risk of MetS and all its constituents.