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. 2017 Apr 4;124(8):915–964. doi: 10.1007/s00702-017-1717-8

Table 2.

MRI findings for differential diagnosis in symptomatic parkinsonism

Entity Typical MRI findings (may vary)
Vascular parkinsonism Lacunar infarctions in the basal ganglia, frontal lobe infarctions, subcortical microangiopathic lesions with diffuse periventicular signal alterations
Normal pressure hydrocephalus Enlargement of lateral cerebral ventricles, ballooning of anterior horn of lateral ventricle, periventricular T2 signal alterations
Toxic-induced parkinsonism
 Manganese Hyperintensities in the globus pallidus, increasing signals in T1-weighted sequences in the striatum and SN
 Ephedron (methcathinone) Increased bilateral and symmetric T1-signal intensity in the globus pallidus and hyperintensities in the SN, no signal abnormalities on T2-weighted images
 Carbon monoxide Transient bilateral symmetric lesions in the globus pallidus with hyperintensities in T2-weighted images
 Cyanide Symmetric hyperintense signal changes in the globus pallidus, putamen, caudate nucleus, and white matter areas in T2-weighted images and FLAIR sequences, lesions in the basal ganglia displaying T1 signal increase with contrast enhancement
 Methanol T2 signal increase and T1 signal decrease in the area of the putamen
Huntington’s disease (Westphal variant) Progressive bilateral atrophy of the striatum and caudate nucleus with enlarged anterior horn of lateral ventricle: in the later course widespread atrophy throughout the cortex;
Wilson’s disease Atrophy of the midbrain, brain stem, and cerebellum; marked T2 hypointensity in the globus pallidus, symmetric T2 hyperintensity in the striatum, lateral thalamus, white matter, and dorsal brain stem; “face of the giant panda”: T2-weighted axial MRI with normal signal at the red nuclei (eyes) and lateral aspects of the SN (ears) with signal increase at the tegmentum and hypointense superior colliculi
Neurodegeneration with iron accumulation (NBIA)
 Panthothenate kinase-associated neurodegeneration (PKAN) Decreased signal in T2-weighted sequences in the globus pallidus, putamen, caudate nucleus, and thalamus; „eye of the tiger“ sign: high signal in the center of the globus pallidus and T2 hypointensity of the surrounding area
 Aceruloplasminaemia and neuroferritinopathy T2 hypointensity in the globus pallidus, SN, striatum, thalamus, and dentate nucleus
Cerebral masses Characteristic structural imaging according to the CNS tumours’ entity
Multiple sclerosis T1‐weighted hypointense lesions (“black holes”) and hyperintensities in T2-weighted sequences in the SN and basal ganglia