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. 2017 May 23;21:206–212. doi: 10.1016/j.ebiom.2017.05.024

Fig. 7.

Fig. 7

a) Vasodilatory effects of nitroglycerin (NTG) (left panel) and nifedipine (NFP) (right panel) on the plateau of contraction induced by potassium chloride (KCl; 10− 1 M) in presence or absence of carfilzomib (CFZ). *p < 0.05 in the comparison between effects of NTG 10− 5 M and NFP 10−7 M. b) Vasodilatory response to nitroglycerin (NTG) (left panel) and nifedipine (NFP) (right panel) on the plateau of contraction induced by noradrenaline (NA; 10− 6 M) in presence or absence of carfilzomib (CFZ). *p < 0.05 in the comparison between the effects of NTG 10− 5 M and NFP 10−7 M on the plateau of contraction induced by NA in presence of CFZ. **p < 0.01 in the comparison between the effects of NTG 10− 5 M and NFP 10−7 M on the plateau of contraction induced by NA in absence of CFZ. c) Vasodilatory effects of nitroglycerin (NTG) (left panel) and nifedipine (NFP) (right panel) on the plateau of contraction induced by angiotensin II (A-II; 10− 5 M) in presence or absence of carfilzomib (CFZ). *p < 0.05 in the comparison between the effects of NTG 10− 5 M and NFP 10−7 M on the plateau of contraction induced by A-II in presence of CFZ. ***p < 0.001 in the comparison between the effects of NTG 10− 5 M and NFP 10−7 M on the plateau of contraction induced by A-II in absence of CFZ.