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. 2017 Jul 17;16:123. doi: 10.1186/s12943-017-0702-z

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — miR-367 mediated the tumor suppressive effects of CASC2 in HCC. a miR-367 restoration reduced the expression of FBXW7 in CASC2-overexpressing MHCC-97H cells. b miR-367 knockdown increased the level of FBXW7 in CASC2-underexpressing Hep-3B cells. c Transwell assays showed that miR-367 could largely reverse the suppressive effects of CASC2 on migration and invasion of MHCC-97H cells. d miR-367 silencing abrogated the pro-metastatic effects of CASC2 knockdown in Hep-3B cells. e miR-367 could largely reverse the suppressive effects of CASC2 on EMT progression of MHCC-97H cells. f miR-367 silencing abolished the promoting effects of CASC2 on EMT progression of Hep-3B cells. *P < 0.05, **P < 0.01