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. 2017 Jun 18;10(6):870–877. doi: 10.18240/ijo.2017.06.07

Figure 4. SE abrogated the activation of NFκB signaling pathway in retinas from STZ-induced diabetic mice.

Figure 4

A: SE reduced the increased expression of phosphorylated NFκBp65 and its subsequent nuclear translocation, representative blots for phosphorylated NFκBp65, nuclear and cytosolic NFκBp65, actin and Lamin B1, and the results represent at least three independent experiments; B: The quantitative densitometric analysis of phosphorylated NFκBp65, and the results are presented as percentage of control (n=3); C: The quantitative densitometric analysis of nuclear and cytosolic NFκBp65, and the results are presented as percentage of control (n=3-4). eP<0.001 compared to control; aP<0.05, fP<0.001 compared to DM.