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. 2017 Jul 18;7:5754. doi: 10.1038/s41598-017-06209-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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p32 heterozygosity increases glucose tolerance and insulin sensitivity. (A) Average fasting blood glucose levels of 52-week-old mice fed NC (n = 6). (B) Glucose tolerance test (GTT) and (C) insulin tolerance test (ITT) in 52-week-old mice fed NC (n = 6). Bar graphs to the right quantify the areas under the curves (AUC). (D) Average fasting blood glucose levels in mice fed an HFD for 10 weeks (WT, n = 7; p32 ^+/−, n = 5). GTT (E) and ITT (F) assays in mice fed an HFD for 10 weeks (n = 5). Bar graphs to the right quantify the areas under the curves (AUC).