Table 1.
Dog ID | Sex | CAD | Genotype top GWAS SNP1 | Represented in figure |
---|---|---|---|---|
1 | Female | Case | Homozygote risk | 1b–d, S1a, S1c, S3b–d |
2 | Male | Case | Homozygote risk | |
3 | Male | Case | Homozygote risk | |
4 | Female | Case | Heterozygote risk | S1b, S1d |
5 | Female | Case | Heterozygote risk | |
6 | Male | Case | Heterozygote risk | |
7 | Female | Case | Heterozygote risk | |
8 | Male | Case | Heterozygote risk | 1a |
9 | Male | Case | Heterozygote risk | |
10 | Male | Control | Homozygote control | 2b |
11 | Female | Control | Homozygote control | 3a, 3b, S6a, S6B |
12 | Male | Control | Homozygote control | S2 |
13 | Male | Control | Homozygote control | 2a, S3a, S4a–c |
14 | Female | Control | Homozygote control |
CAD canine atopic dermatitis, GWAS genome‐wide association study, SNP single nucleotide polymorphism.