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. Author manuscript; available in PMC: 2018 Jun 1.
Published in final edited form as: Alcohol. 2017 May 18;61:51–61. doi: 10.1016/j.alcohol.2017.01.014

Fig. 3. Ethanol-induced dilation of in vitro pressurized fetal MCA.

Fig. 3

A. Trace showing fetal cerebral artery dilation in response to 63-mg/dL ethanol. B. Bar graph displays averaged data: ethanol-induced dilation of fetal cerebral artery vanishes in presence of CB1 and CB2 receptor blockers AM251 and AM630, respectively. Data from control- and alcohol-exposed fetuses were compared using independent unpaired t tests. *Significantly different from data in absence of CBR blockers, p < 0.05. (n/N) = number of artery segments/number of fetuses.