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. 2017 Jul 19;7:5918. doi: 10.1038/s41598-017-06368-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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FPR1 activation accelerated the migration of SW480. The motility of SW480 (a,b) and HT29 (c,d) in wound-healing model was assayed in the presence of fMLF (1 μM) or 10% FBS with or without a 15 min pretreatment with cyclosporine H (1 μM). Then cells were photographed at 24 h and 48 h. *p < 0.05 compared with control, ^# p < 0.05 compared with the cells treated with fMLF. All the data shown are mean ± SEM from three separate experiments, each in triplicates.