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. 2017 Jul 19;7:5925. doi: 10.1038/s41598-017-06132-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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CEP55 expression was clinically correlated with the activation of nuclear factor kappaB (NF-κB) signalling in patients with pancreatic cancer (PANC). (a) Changes in mRNA expression of NF-κB-regulated genes (MYC, CCND1, BCLXL, MMP9, TWIST, VEGFC and IL6) in nine pancreatic cancer tissues assessed by real-time PCR. (b) Western blotting analysis of CEP55 expression in these nine pancreatic cancer tissues (Upper panel). The endogenous NF-κB activity in these nine pancreatic cancer tissues detected by an electrophoretic mobility shift assay (EMSA) (Lower panel). (c) The Spearman correlation coefficient was calculated to assess the significance of association between CEP55 protein and NF-κB activity. *P < 0.05.