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. 2017 Jul 19;7:5824. doi: 10.1038/s41598-017-05163-w

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of RARα agonist on ApoC-III and TGs production in high fat diet induced fatty liver mice. 4-month-old HFD mice treated daily with AM580 at 1 and 5 mg/kg by oral gavage. After 9 days, mice were sacrificed for plasma and tissue level analysis of ApoC-III and TGs and TCs. (a) Percentage body weight change; (b) Liver weight; (c) Hepatic lipids were stained by oil red O staining; (d) TGs levels in liver lysate and (e) Plasma TGs levels. (f) Plasma ApoC-III levels were measured by ELISA assay; (g) Visceral adipose tissue weights and (h) Plasma total cholesterol levels were measured by biochemical assay after 9 days of AM580 treatment in DIO mice. Data are means ± S.E. of 5–8 animals per group. *P < 0.05 versus vehicle.