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. 2017 Jul 19;16:125. doi: 10.1186/s12943-017-0676-x

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — ARHGAP11A expression contributes to the metastatic phenotype of HCC. a-d Transwell assays on miR19a-treated or ARHGAP11A-silenced HCCLM3 cells. e and f The expression levels of ARHGAP11A in cancerous and paired adjacent non-cancerous tissues in the non-metastatic and metastatic groups. g The expression levels of ARHGAP11A in non-metastatic and metastatic HCC tissues. h The correlation between HOXD-AS1 transcript levels and ARHGAP11A mRNA levels was measured in 44 HCC tissues. The dCt values (normalized to GAPDH) were subjected to Pearson correlation analysis. i and j The prognostic significance of ARHGAP11A in HCC patients. The Caner Genome Atlas project (TCGA) dataset was analyzed to reveal the effect of ARHGAP11A mRNA levels on the overall survival (OS) or disease-free survival (DFS) of patients with HCC using cBioPortal (http://cbioportal.org)