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. Author manuscript; available in PMC: 2018 Jul 1.
Published in final edited form as: Hypertension. 2017 May 15;70(1):158–165. doi: 10.1161/HYPERTENSIONAHA.117.09279

Table 3.

Associations between brachial pulse pressure and CMR-derived measures in participants 18 to 64 years of age (n=2,001)

CMR-derived measures β (SE) P-value Model R2, %
Mean arterial pressure 5.5 (0.2) <0.0001 54.3
Aortic arch PWV 2.4 (1.7) <0.0001
Aortic size −1.1 (1.1) <0.0001
Left ventricular stroke volume 1.8 (0.2) <0.0001

β = unstandardized regression coefficient; model R2 = a measure for the model prediction. The table shows adjusted β coefficient (SE) associated with 1 SD increases of each exposure. The one-SD increment of each exposure is as follows: mean arterial pressure, 11.2 mmHg; aortic arch PWV, 2.6 m/s; aortic size, 180.6 mm2; left ventricular stroke volume, 16.2 ml. Mean arterial pressure, aortic arch PWV, aortic size, and left ventricular stroke volume entered into the same model, and the following variables were used as covariates: demographic variables (age, sex, and race) + clinical characteristics (height, weight, heart rate, smoking, physical activity, total cholesterol, fasting glucose, and eGFR) +VAT. Statistical significance was defined as P <0.05.