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. 2017 Jul 20;7:6024. doi: 10.1038/s41598-017-06243-7

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Disruption of HDAC9 translocation and MEF2 interaction affect TC axon branching. Representative tracings of control and mutant HDAC9 transfected axons. Thalamic cells were electroporated with only mCherry to observe control TC axon branching. To examine the effect of the HDAC9 mutants, mCherry was co-transfected with either of HDAC9, nucHDAC9, or nucHDAC9ΔMEF2.